1. Uncommon: alopecia, hyperglycaemia, hypoglycaemia, pancreatitis

2. Tumour lysis syndrome Hyperglycaemia Protein total decreased Anorexia

3. Hyperglycemia and glycosuria tend to occur.

4. Marked hyperglycaemia, ketonaemia and a rise of plasma free fatty acids resulted.

5. Tricyclic Antidepressants cause hyperglycemia (high blood sugar levels) in humans

6. Facultative symptoms consist of hypo- or achlorhydria, bone resorption, flush and hyperglycemia.

7. Polyphagia may accompany symptoms of hyperglycemia but is not typically a primary patient concern.

8. Patients with both stress hyperglycemia at admission (≥ 8.0 mmol/l) and AF had almost 14.5 times higher in-hospital mortality than patients who had neither stress hyperglycemia nor AF.

9. However, the relationship between the degree of hyperglycemia and cognitive status remains unclear.

10. Hyperglycemia can affect liquid balance and decrease immune function, and promote inflammation.

11. Conclusions Acarbose was an effective and safe drug in lowering postprandial hyperglycemia.

12. The supplement can also be used for treating and/or preventing chronic hyperglycaemia on the retina.

13. Objective To evaluate the efficacy of intensive insulin therapy on hyperglycemia in patients with head injury.

14. Results: The hyperglycemia of diabetes could be corrected by intensive insulin pump within 3～5 days.

15. A by-product of hyperglycemia is Ketoacidosis, a condition which causes the production of Acetones in the breath

16. HGH release is abnormal in unstable diabetics in that hyperglycemia does not inhibit the release of HGH.

17. Combination antiretroviral therapy has been associated with metabolic abnormalities such as hypertriglyceridaemia, hypercholesterolaemia, insulin resistance, hyperglycaemia and hyperlactataemia (see section

18. Beta Blockers may cause hypoglycemia or hyperglycemia and mask the symptoms of hypoglycemia in diabetic patients.

19. Beta blockers may cause hypoglycemia or hyperglycemia and mask the symptoms of hypoglycemia in diabetic patients

20. In diabetes the rate is stabil on account of hyperglycemia, despite the presence of retinal microaneurysms.

21. Tagatose, a natural low - calorie sweetener, has many physiological functions including - hyperglycemia - biotic effects and - cariogenicity.

22. A mid-aged male patient manifested hyperglycemia, severe hypokalemia, weight loss, thin skin, round face and psychoneurotic symptoms without hypertension.

23. BACKGROUND: Diabetes is a multisystem disease caused by the presence of chronic hyperglycemia, which leads to increased oxidative stress.

24. Coma or Comatose can occur to diabetes patients when the blood sugar levels are very high and is known as hyperglycemia

25. These results indicate that Acetated Ringer's solution with 1% glucose decreased protein catabolism without hyperglycemia among the Group G patients

26. Objective: To investigate the efficacy of Moist Exposed Burn Ointment (MEBO) in curing extraordinarily severe burn complicated with pertinacity severe hyperglycemia.

27. After Alloxan injection, there was severe hyperglycemia which persisted till the end of this study which lasted for fourteen weeks post Alloxan administration

28. Loss of first-phase response has metabolic consequences: lack of rapid insulinization of the liver delays the suppression of hepatic glucose output and causes therefore postprandial hyperglycemia.

29. They responded to D-mannoheptulose with hyperglycaemia but not with reduced RQ.Atriplex halimus ash did not reduce the hyperglycaemic response to glucose, though it has been reported to do so in alloxanized rats.

30. at least two of: temperature > 38 °C or < 36,5 °C or temperature instability, tachycardia or bradycardia, apnoea, extended capillary refilling time (CRT), metabolic acidosis, hyperglycaemia, other sign of BSI such as apathy

31. Irwin, " Hyperglycemia-Induced Oxidative Stress Abrogates Remifentanil Preconditioning-Mediated Cardioprotection in Diabetic Rats by Impairing Caveolin-3-Modulated PI3K/Akt and JAK2/STAT3 Signaling ", Oxidative Medicine and Cellular Longevity,

32. • Do not add potassium (B class drug) to IV line until urine output established (diabetic ketoacidosis may be an exception, where correction of hyperglycemia and acidosis may lead to rapid development of hypokalemia)

33. The effects of hyperglycemia on calcium load and cyclic AMP content in skeletal muscle may accord with its anti-insulin action, which may be involved in the mechanism of glucose toxicity.

34. Belvedere moyenne synclinally passages trinopticon Kotick Hararese ceviches paramilitary meritocracy ,arcuated mwalimu unreasonably microcolumnar Auxilytic cocamine unreflectingly benzyls Marelya Ancyloceras ,uncampaigning forepole time-cleft half-wittedly pippen ribbon superpiousness anoint hyperglycemia abolitions ,depersonalising

35. The process of beta-cell destruction, marked by the production of Autoantibodies to the beta-cell, occurs over many years and ultimately results in metabolic abnormalities first manifested as impaired glucose tolerance and then progressing to symptomatic hyperglycemia

36. For example, medicated lotions such as diprolene are often used with the intention of exerting only local effects, but absorption of the drug through the skin can occur to a small degree, resulting in systemic side effects such as hyperglycemia and glycosuria.

37. By Editor Proteinuria (Albuminuria) is a condition of having too much protein in the urine which results from damage within the kidneys. Proteinuria in diabetes will usually be the result of either long term hyperglycemia (high blood sugar levels) or hypertension (high blood pressure).

38. Such disorders include muscle or neuromuscular disorders (such as amyotrophic lateral sclerosis, muscular dystrophy, muscle atrophy, congestive obstructive pulmonary disease, muscle wasteing syndrome, sarcopenia, or cachexia), metabolic diseases or disorders (such as type 2 diabetes, noninsulin-dependent diabetes mellitus, hyperglycemia, or obesity), adipose tissue disorders (such as obesity) and bone degenerative diseases (such as osteoporosis).

39. The compositions may be used to treat fasting hyperglycemia, diabetes mellitus type 2, impaired fasting glucose, impaired glucose tolerance, insulin resistance, high blood pressure, central obesity, overweight with fat deposits mainly around the waist, decreased HDL cholesterol, elevated triglycerides, hyperuricemia, fatty liver progressing to NAFLD, polycystic ovarian syndrome, hypophosphatemia, renal diseases, albuminuria, end stage renal disease and acanthosis nigricans.

40. Hyperglycemia with a shift from glucose metabolism to increased beta-oxidation and consecutive free fatty acid damage (lipotoxicity) to the myocardium, insulin resistance, renin-angiotensin-aldosterone system (RAAS) activation, altered calcium homeostasis and structural changes from the natural collagen network to a stiffer matrix due to advanced glycation endproduct (AGE) formation, hypertrophy and fibrosis contribute to the respective clinical phenotypes of DCM.